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HYPOCALCEMIA = ionized Ca < 2.0mEq/L Etiology:
- During trauma, shock, sepsis, fat embolism Ca tends to move into the cell
- Pancreatitis - fatty acids combine with Ca and form soaps.
- Drugs (cimetidine, phosphate containing enemas)
- HypoPTH
- Renal Failure 2ry to production of 125-(OH)-vitamin D. Also 2ry to phosphate.
- HypoMg
- vitamin D
- Respiratory alkalosis (decreases ionized Ca)
What is important is the amount of ionized Ca and not the total amount of Ca. As a matter of fact during hypoalbuminemic state total Ca is low but ionized Ca is normal. S & S :
- Weakness, N/V, fatigue
- CNS -tetany,
Chvosteck's, Trousseau's, perioral numbness, psychosis.
CV -heart failure, dysrhythmia
ENDOCRIN - increase in PTH (negative feedback)
DX :
- S&S are helpful
- EKG shows > QT, possible TW inversion
- BUN, Mg, PO4,
Treatment:
- If pt shows no S&S, give oral Ca
- If symptoms present, give Ca gluconate (10ml amp contains 4.6mEq/L of Ca) or CaCl (10ml amp contains 13mEq/L Ca). Both are given IV over 20-30min.
- If albumin or Mg are low, supplementing these may suffice
- Due to prolonged QT and possibility of V.Tach pts should be on monitor
and admitted to monitored bed. Hypercalcemia conversely gives shortening in QT which is not associated with arrhythmias
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