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HYPERKALEMIA
Etiology:
- Renal Failure, Acidosis, Cell lysis (burn, chemotherapy), Potassium sparing meds, steroids.,
b-blockers, Insulin
deficiency, Thrombo/Leukocytosis, ACE-i, Tubular unresponsiveness (SLE, MM, SC anemia)
S & S :
- Neurologic (weakness, reflexes, paresthesias) and Cardiac (PVC's, bradycardia, asystole).
DX :
- K+ = 5.6-6.0 on EKG T wave in precordial leads
- K+ = 6.0-6.5 on EKG >PR and >QT
- K+ = 6.5- 7.0 on EKG flattening of P wave
- K+ >7.5 on EKG BBB pattern, >QRS
- K+ >10 on EKG sine wave (looks almost as if it was A-line registration wave)
NOTE!!! This classification doesn't apply to pts who are used to hyperkalemic state such as "renal" pts and not infrequently pt will
have only high T wave corresponding to K of 6.0, yet the lab report may be higher. Death occurs in diastole. Treatment:
- K+ level <6.0
-produce diuresis.
- K+ level >6.0
- give kayexalate 20-30gm. This binds K+ and it works when pt has bowel movement. Each gram binds 1mEq of K, but high Na concentration may give volume overload.
- K+ level >6.5
- give RI 10 U + D 50 Glucose. This
works by redistribution. Onset of action is in 1/2hr. Action duration 3hrs
K+ level 6.5-7.0 -add NaHCO3 50-100mEq over 5 min. Works by redistribution and antagonism. Onset of action in 10min. Action duration 1-2hrs.
K+ level >7.0-7.5 give the following sequence: CaCl (or Ca Gluconate) - NaHCO - D50+RI 10 U. CaCl ( or CaGluconate) give 10-20ml over 5 min.
Works by antagonism. Onset of action 1-3min. Duration of action ½ hr. CaGluconate contains 5mEq of Ca per ampule while CaCl - 13.5mEq per ampule. Also Ca is faster available from Chloride form. If hyperK
is associated with DIG toxicity Ca is not recommended. Note! The sequence is: CaCl-NaHCO3-D50+RI+diuresis
Patient that is dialysis dependent, adding Albuterol (Ventolin, Proventil ) via neb (in addition to above described treatment modalities), was shown to lower the K level.
Hemodialysis, especially if RF.
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