ATRIAL FLUTTER
Etiology: 1.
CAD, MI, TSH, PE, COPD, RH, CHF, ASD. As compare to A.fib, the A.flut is not seen in normal heart.
S & S: 1. Pt may experience palpitations/tachycardia only or may have more unstable vital signs (e.g. CHF).
2. Pt may c/o chest pain due to ischemia induced by tachycardia or dyspnea if CHF occurredDX:
- Atrial rate 250-350 b/min
- "sawtooth" pattern in II, III, aVF.
Atrial-to-Ventricle conduction may vary (1:1, 2:1, 3:1). The 1:1 block may be hard to distinguish from VF. The 2:1 block will present with HR
of 150 or somewhere very close to it (145, 140). A.flut with 3:1 block will have ventricular rate of 100 b/min.
IfA.flut presents with variable block it may be difficult to distinguish from A.fib. Additionally, these two rhythms may coexist leading to common presentation of "Fib-Flut"
Treatment:
1. Hemodynamically stable
- dig, CCB or -blockers as in case of A.fib (see above) are given to achieve control and to prevent rapid ventricular response. Carotid massage can be tried.
2. Hemodynamically unstable or having S & S of ischemia, SOB, MI – sync. cardioversion with 25-50 J is tried initially and then 100-200-300J. 3.
Although the risk of thromboembolism is less than in case of A.fib, the risk is nonetheless higher than in pts with NSR. Thus, same approach as in case of A.fib (i.e. cardioversion if the duration is less than 48 hrs or
anticoagulation for 3-4 wks if more than 48hrs) is recommended. |